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(2018), Cell Metab 27(3), PubMed 29514064 Statistics from preclinical literature NAD+ (nicotinamide adenine dinucleotide), a key cofactor of redox reactions and substrate for sirtuins, PARP, CD38 , molecular weight 663.43 g/mol (not a peptide, but traditionally included in peptide research collections) Identified by Arthur Harden (Nobel Prize 1929) in 1906 as a “coferment” Standard experimental intravenous dose in clinical studies: 250–750 mg/day for 4–10 days (Grant 2019, Conlon & Bird 2020) NAD+ levels decline with age by ~50 % in various tissues between ages 30 and 80 (Massudi 2012) Mechanism of supplementation: direct intravenous repletion, or precursors NR (nicotinamide riboside), NMN (nicotinamide mononucleotide) that are converted to NAD+ via NAMPT/NRK enzymes In Conlon & Bird (2020, n=11): NAD+ IV infusion 750 mg/day for 6 days raised plasma NAD+ by ~40 % over baseline NR and NMN are registered in the USA as dietary supplements (NDI status)
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Size:4 Bottles - £15.19 / a bottle
For example, according to a recent review by researchers in the US looking at peptides often marketed to patients with musculoskeletal injuries, BPC-157 “demonstrated potential benefits in tendon and muscle repair, but these findings are largely unvalidated in human trials”, with no randomised control trials in humans relating to such uses, and the only case series having serious flaws
